5.1 Gastric Residence

Gastric residence defines how long food remains in the stomach before gastric release into the small intestine. In Feline FBM it affects the concentration and timing of fat entry into downstream digestive handling.

Gastric residence is not the same as disease-level gastric stasis. It is a control variable in the digestion and fat transport layer, read from upstream food structure—not from stool form alone.

Slower release can change fat entry concentration and timing for Bile Salt Dispersion and pancreatic enzyme processing. All backtrace returns to upstream food structure.

When and how concentrated fat enters the small intestine.

Upstream structure sets residence pattern before clinical labels apply.

Fat Processing Rate must match entry timing downstream.

Fat-dominant structure increases relevance of gastric release pacing.

upstream food structure

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gastric residence and gastric release

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fat entry concentration and timing

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bile salt dispersion and pancreatic enzyme processing

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fat processing rate and observable outputs

Loose stool, reduced palatability feedback, or digestive discomfort may appear when release timing mismatches downstream handling. These are backtrace entry points, not root causes.