6 Water Recovery and Stool System · 6.6 Constipation and Loose Stool Pathways
6.6 Constipation and Loose Stool Pathways
Feline Fat-Based Metabolism (Fat-Based Metabolism)
Constipation and loose stool are opposite output patterns along stool water content, water recovery, secretion, and propulsion variables. They should not be treated as isolated diseases by default.
This page sits in the water recovery and stool system layer. Both patterns backtrace to upstream food structure—not to drinking water alone or residue shortage alone.
constipation index (stool water decreases / output resistance increases)
stool water content · colonic propulsion · proximal water recovery
SGLT1-Mediated Water-Sodium Absorption and related variables
upstream food structure
loose stool index (stool water increases / formation decreases)
colonic secretion · colonic propulsion · absorption variables
Bile-Acid-Related Colonic Regulation · Fat Processing Rate
upstream food structure
Stool water content, proximal small-intestinal water recovery, colonic propulsion, upstream food structure.
SGLT1-mediated water-sodium absorption, slower propulsion, increased recovery before output.
Colonic secretion, bile-acid-related colonic regulation, fat processing rate mismatch.
Constipation and loose stool indices are observable outputs and backtrace entry points—not root causes.
Opposite patterns share variables but weight different segments of the chain.
both patterns explained only by drinking water
constipation = fiber residue shortage by default
loose stool = bile acid single cause
isolated disease labels without structure backtrace
obstruction, severe pain, bloody stool, or protracted vomiting → clinical boundary
This page defines dual pathway structure only. Not a clinical diagnosis system, not a treatment protocol, not an effect-guarantee system.